Ben Laufer
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    • TMEM106B reduction does not rescue GRN deficiency in iPSC-derived human microglia and mouse models
    • Lipid nanoparticle delivery limits antisense oligonucleotide activity and cellular distribution in the brain after intracerebroventricular injection
    • Cross-species transcriptomic atlas of dorsal root ganglia reveals species-specific programs for sensory function
    • Multi-omic brain and behavioral correlates of cell-free fetal DNA methylation in macaque maternal obesity models
    • Disease-associated oligodendrocyte responses across neurodegenerative diseases
    • Elevated exposures to persistent endocrine disrupting compounds impact the sperm methylome in regions associated with autism spectrum disorder
    • Placenta and fetal brain share a neurodevelopmental disorder DNA methylation profile in a mouse model of prenatal PCB exposure
    • Placental methylome reveals a 22q13.33 brain regulatory gene locus associated with autism
    • Long-term effects of wildfire smoke exposure during early life on the nasal epigenome in rhesus macaques
    • Epigenetic Impacts of Early Life Stress in Fetal Alcohol Spectrum Disorders Shape the Neurodevelopmental Continuum
    • Stable DNMT3L overexpression in SH-SY5Y neurons recreates a facet of the genome-wide Down syndrome DNA methylation signature
    • Cord blood DNA methylome in newborns later diagnosed with autism spectrum disorder reflects early dysregulation of neurodevelopmental and X-linked genes
    • Low-pass whole genome bisulfite sequencing of neonatal dried blood spots identifies a role for RUNX1 in down syndrome DNA methylation profiles
    • Whole genome bisulfite sequencing of Down syndrome brain reveals regional DNA hypermethylation and novel disorder insights
    • Epigenomic Convergence of Neural-Immune Risk Factors in Neurodevelopmental Disorder Cortex
    • Imprinting effects of UBE3A loss on synaptic gene networks and Wnt signaling pathways
    • Snord116-dependent diurnal rhythm of DNA methylation in mouse cortex
    • Experience-dependent neuroplasticity of the developing hypothalamus: integrative epigenomic approaches
    • Changes to histone modifications following prenatal alcohol exposure: An emerging picture
    • Long-term alterations to DNA methylation as a biomarker of prenatal alcohol exposure: From mouse models to human children with fetal alcohol spectrum disorders
    • A Long-Term Neuroepigenomic Profile of Prenatal Alcohol Exposure
    • Alteration of gene expression, DNA methylation, and histone methylation in free radical scavenging networks in adult mouse hippocampus following fetal alcohol exposure
    • Strategies for precision modulation of gene expression by epigenome editing: An overview
    • Associative DNA methylation changes in children with prenatal alcohol exposure
    • DNA methylation differences in monozygotic twin pairs discordant for schizophrenia identifies psychosis related genes and networks
    • DNA methylation in psychosis: Insights into etiology and treatment
    • Fetal alcohol and the right to be born healthy
    • Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: A model for fetal alcohol spectrum disorders
    • Molecular changes during neurodevelopment following second-trimester binge ethanol exposure in a mouse model of fetal alcohol spectrum disorder: From immediate effects to long-term adaptation
    • The effects of olanzapine on genome-wide DNA methylation in the hippocampus and cerebellum
    • Third trimester-equivalent ethanol exposure is characterized by an acute cellular stress response and an ontogenetic disruption of genes critical for synaptic establishment and function in mice
    • Neurodevelopmental epigenetic etiologies: Insights from studies on mouse models of fetal alcohol spectrum disorders
    • Long-lasting alterations to DNA methylation and ncRNAs could underlie the effects of fetal alcohol exposure in mice
    • Selected rapporteur summaries from the XX world congress of psychiatric genetics, Hamburg, Germany, october 14-18, 2012
    • Olanzapine induced DNA methylation changes support the dopamine hypothesis of psychosis
    • Reduced expression of brain cannabinoid receptor 1 (Cnr1) is coupled with an increased complementary micro-RNA (miR-26b) in a mouse model of fetal alcohol spectrum disorders
    • A Macro Role for Imprinted Clusters of MicroRNAs in the Brain
    • Long-term alterations to the brain transcriptome in a maternal voluntary consumption model of fetal alcohol spectrum disorders

Fetal alcohol and the right to be born healthy

Jan 1, 2014·
Shiva M. Singh
,
Benjamin I. Laufer
,
Joachim Kapalanga
· 0 min read
DOI PDF
Type
2
Publication
Frontiers in Genetics
Last updated on Jul 24, 2021
← DNA methylation in psychosis: Insights into etiology and treatment Jan 1, 2015
Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: A model for fetal alcohol spectrum disorders Jan 1, 2014 →

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