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Neurodevelopmental epigenetic etiologies: Insights from studies on mouse models of fetal alcohol spectrum disorders
Benjamin I. Laufer, Eric J. Diehl, Shiva M. Singh
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Long-lasting alterations to DNA methylation and ncRNAs could underlie the effects of fetal alcohol exposure in mice
Fetal alcohol spectrum disorders (FASDs) are characterized by life-long changes in gene expression, neurodevelopment and behavior. What …
Benjamin I. Laufer, Katarzyna Mantha, Morgan L. Kleiber, Eric J. Diehl, Sean M.F. Addison, Shiva M. Singh
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Olanzapine induced DNA methylation changes support the dopamine hypothesis of psychosis
BACKGROUND: The dopamine (DA) hypothesis of schizophrenia proposes the mental illness is caused by excessive transmission of dopamine …
Melkaye G. Melka, Christina A. Castellani, Benjamin I. Laufer, Raj N. Rajakumar, Richard O'Reilly, Shiva M. Singh
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Reduced expression of brain cannabinoid receptor 1 (Cnr1) is coupled with an increased complementary micro-RNA (miR-26b) in a mouse model of fetal alcohol spectrum disorders
Background: Prenatal alcohol exposure is known to result in fetal alcohol spectrum disorders, a continuum of physiological, …
Randa L. Stringer, Benjamin I. Laufer, Morgan L. Kleiber, Shiva M. Singh
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A Macro Role for Imprinted Clusters of MicroRNAs in the Brain
Evidence suggests that $∼$95% of the human genome may produce noncoding RNAs (ncRNAs). Approximately 30% of parentally imprinted …
Benjamin I. Laufer, Shiva M. Singh
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Long-term alterations to the brain transcriptome in a maternal voluntary consumption model of fetal alcohol spectrum disorders
Many women continue to consume low to moderate quantities of alcohol during pregnancy, which can result in the variable …
Morgan L. Kleiber, Benjamin I. Laufer, Elise Wright, Eric J. Diehl, Shiva M. Singh
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